<efrbr:recordSet xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xmlns:efrbr="http://vfrbr.info/efrbr/1.1" xmlns:efrbr-work="http://vfrbr.info/efrbr/1.1/work" xmlns:efrbr-expression="http://vfrbr.info/efrbr/1.1/expression" xmlns:efrbr-manifestation="http://vfrbr.info/efrbr/1.1/manifestation" xmlns:efrbr-person="http://vfrbr.info/efrbr/1.1/person" xmlns:efrbr-corporateBody="http://vfrbr.info/efrbr/1.1/corporateBody" xmlns:efrbr-concept="http://vfrbr.info/efrbr/1.1/concept" xmlns:efrbr-structure="http://vfrbr.info/efrbr/1.1/structure" xmlns:efrbr-responsible="http://vfrbr.info/efrbr/1.1/responsible" xmlns:efrbr-subject="http://vfrbr.info/efrbr/1.1/subject" xmlns:efrbr-other="http://vfrbr.info/efrbr/1.1/other" xsi:schemaLocation="http://vfrbr.info/efrbr/1.1 http://vfrbr.info/schemas/1.1/efrbr.xsd"><efrbr:entities><efrbr-work:work identifier="http://purl.tuc.gr/dl/dias/F2DCDF11-38E7-485D-B76D-ED2712E0D888"><efrbr-work:titleOfTheWork>Involvement of tgf-a, rock1, dock2 and caveolin-1 in osteoarthritis pathogenesis</efrbr-work:titleOfTheWork></efrbr-work:work><efrbr-expression:expression identifier="http://purl.tuc.gr/dl/dias/F2DCDF11-38E7-485D-B76D-ED2712E0D888"><efrbr-expression:titleOfTheExpression>Involvement of tgf-a, rock1, dock2 and caveolin-1 in osteoarthritis pathogenesis</efrbr-expression:titleOfTheExpression><efrbr-expression:formOfExpression vocabulary="DIAS:TYPES">
            Peer-Reviewed Journal Publication
            Δημοσίευση σε Περιοδικό με Κριτές
         </efrbr-expression:formOfExpression><efrbr-expression:dateOfExpression type="issued">2015-10-25</efrbr-expression:dateOfExpression><efrbr-expression:dateOfExpression type="published">2014</efrbr-expression:dateOfExpression><efrbr-expression:languageOfExpression vocabulary="iso639-1">en</efrbr-expression:languageOfExpression><efrbr-expression:summarizationOfContent>Osteoarthritis (OA) is a debilitating disease of the joints characterized by cartilage degradation. To date there is no available pharmacological treatment to inhibit disease progression neither is there a suitable biomarker for early disease prognosis. The aim of the present study was to investigate the molecular pathways involved between four identified OA susceptibility genes and their role in OA pathogenesis. 
</efrbr-expression:summarizationOfContent><efrbr-expression:useRestrictionsOnTheExpression type="creative-commons">http://creativecommons.org/licenses/by/4.0/</efrbr-expression:useRestrictionsOnTheExpression><efrbr-expression:note type="journal name">Osteoarthritis and Cartilage</efrbr-expression:note><efrbr-expression:note type="journal number">22</efrbr-expression:note></efrbr-expression:expression><efrbr-person:person identifier="http://users.isc.tuc.gr/~mzervakis"><efrbr-person:nameOfPerson vocabulary="TUC:LDAP">
            Zervakis Michalis
            Ζερβακης Μιχαλης
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            D. Kafetzopoulos
         </efrbr-person:nameOfPerson></efrbr-person:person><efrbr-person:person identifier="http://users.isc.tuc.gr/~kkalantzaki"><efrbr-person:nameOfPerson vocabulary="TUC:LDAP">
            Kalantzaki Kalliopi
            Καλαντζακη Καλλιοπη
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            E. Chatziliadou
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            S. Dubos
         </efrbr-person:nameOfPerson></efrbr-person:person><efrbr-corporateBody:corporateBody identifier="http://www.elsevier.com/"><efrbr-corporateBody:nameOfTheCorporateBody vocabulary="S/R:PUBLISHERS">
            Elsevier
         </efrbr-corporateBody:nameOfTheCorporateBody></efrbr-corporateBody:corporateBody><efrbr-concept:concept identifier="F1998E91-B4B4-4336-87B1-CA6596E89493"><efrbr-concept:termForTheConcept>
            Osteoarthritis
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